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ALT Reference COVID Statistics from High-Quality Data Sources Compiled by the Pandemic Mitigation Collaborative (PMC19.com), Jul 12, 2024 % of Population Actively Infectious Chances Someone is Infectious… In a Group of 10 In a Group of 30 In a Group of 50 Primary Data Source Reference Last Updated U.S.A. 1.37% (1 in 73) 12.9% 33.9% 49.9% Wastewater "Michael Hoerger, PhD, MSCR, MBA @michael_hoerger pmc19.com/data" July 12, 2024 Canada 1.37% (1 in 73) 12.9% 33.9% 49.8% Wastewater "Tara Moriarty, PhD @MoriartyLab covid19resources.ca" July 7, 2024
![A heterogeneous set of hypotheses have been proposed to explain the pathophysiology of long COVID involving viral factors, host factors, and tissue damage.[6] Prominent causal hypotheses include the following: (1) viral persistence, either of infectious virions, viral RNA or viral proteins [7] [8]; (2) reactivation of latent viruses[9]; (3) immune dysregulation and autoimmunity triggered by the infection [6]; and (4) chronic tissue and organ damage characterized by thrombotic endothelialitis, endothelial inflammation, hyperactivated platelets, and fibrinaloid microclots.[10]](https://pbs.twimg.com/media/GRCfHXDXoAAXia_.jpg?name=small&format=webp)
ALT A heterogeneous set of hypotheses have been proposed to explain the pathophysiology of long COVID involving viral factors, host factors, and tissue damage.[6] Prominent causal hypotheses include the following: (1) viral persistence, either of infectious virions, viral RNA or viral proteins [7] [8]; (2) reactivation of latent viruses[9]; (3) immune dysregulation and autoimmunity triggered by the infection [6]; and (4) chronic tissue and organ damage characterized by thrombotic endothelialitis, endothelial inflammation, hyperactivated platelets, and fibrinaloid microclots.[10]
































